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Through these future conversations, participants become aware of the potential of possible, probable, or alternative futures, and explore how they might influence organisational change and contribute to the creation of strategies that pave the way for desired change.

Everyone interested in sharing their thinking and practice vis-à-vis new workplace learning in formal and non-formal learning settings from the workplace-learning sector, education institutions, the policy sector, and governmental organisations

Tools that will be explored include scenario thinking, future wheels, causal layered analysis, horizon scanning, future artefacts, and ‘what-if’ scenarios.

A4 Afternoon Event

Time: Price: 95.00 € Status: places available

Complete workshop description

Date Wednesday, Dec 5 Time Price: 95.00 € Status: places available

Iuliia Shnai

Lappeenranta University of Technology, Finland

I obtained Bachelor Degree majoring in Management of Innovation Technologies in Peter the Great St. Petersburg Polytechnic University (SPbSTU). Following that, I attended a Double Degree Program between Peter the Great St. Petersburg Polytechnic University (SPbSTU) and Lappeenranta University of Technology (LUT). Main field of the Program was Industrial Engineering and Project Management. Currently I am 3rd year doctoral student in the LUT School of Engineering. In my research,I focus on the one of the blended learning concepts flipped classroom. Practically, I provide experiments with transition courses from traditional to flipped classroom and fullyonline format. I am interested in new education technologies and innovative learning designs and teaching approaches.


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Arnob Khan

Lappeenranta University of Technology, Finland

This Pre-Conference Workshop, which is structured as a flipped event, introduces you to flipped classroom design through open educational resources and provides practical support to develop and apply new flipped-classroom design skills.

Using open online sources is a cost-effective approach that can lead to positive learning outcomes. We will provide you with an overview of existing free online tools and their most effective implementation in your future flipped classroom.

These tools will include:

As a flipped Workshop, you will have the opportunity to familiarise yourself with materials before OEB; our time at the conference will be devoted primarily to practical activities. The actual tools with which we will experiment at OEB are to be selected based on your opinions and preferences gathered prior to the conference.

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Zimmers, Teresa A; Jin, Xiaoling; Zhang, Zongxiu; Jiang, Yanlin; Koniaris, Leonidas G


Hepatic steatosis is a common histological finding in obese patients. Even mild steatosis is associated with delayed hepatic regeneration and poor outcomes following liver resection or transplantation. We sought to identify and target molecular pathways that mediate this dysfunction. Lean mice and mice made obese through feeding of a high-fat, hypercaloric diet underwent 70 or 80% hepatectomy. After 70% resection, obese mice demonstrated 100% survival but experienced increased liver injury, reduced energy stores, reduced mitoses, increased necroapoptosis, and delayed recovery of liver mass. Increasing liver resection to 80% was associated with mortality of 40% in lean and 80% in obese mice ( P < 0.05). Gene expression profiling showed decreased epidermal growth factor receptor (EGFR) in fatty liver . Meta-analysis of expression studies in mice, rats , and patients also demonstrated reduction of EGFR in fatty liver . In mice, both EGFR and phosphorylated EGFR decreased with increasing percent body fat. Hydrodynamic transfection of EGFR plasmids in mice corrected fatty liver regeneration , reducing liver injury, increasing proliferation, and improving survival after 80% resection. Loss of EGFR expression is rate limiting for liver regeneration in obesity. Therapies directed at increasing EGFR in steatosis might promote liver regeneration and survival following hepatic resection or transplantation. Copyright © 2017 the American Physiological Society.

Hyperplasia vs hypertrophy in tissue regeneration after extensive liver resection.

Marongiu, Fabio; Marongiu, Michela; Contini, Antonella; Serra, Monica; Cadoni, Erika; Murgia, Riccardo; Laconi, Ezio


To address to what extent hypertrophy and hyperplasia contribute to liver mass restoration after major tissue loss. The ability of the liver to regenerate is remarkable on both clinical and biological grounds. Basic mechanisms underlying this process have been intensively investigated. However, it is still debated to what extent hypertrophy and hyperplasia contribute to liver mass restoration after major tissue loss. We addressed this issue using a genetically tagged system. We were able to follow the fate of single transplanted hepatocytes during the regenerative response elicited by 2/3 partial surgical hepatectomy (PH) in rats . Clusters of transplanted cells were 3D reconstructed and their size distribution was evaluated over time after PH. Liver size and liver DNA content were largely recovered 10 d post-PH, as expected ( e.g ., total DNA/ liver /100 g b.w. was 6.37 ± 0.21 before PH and returned to 6.10 ± 0.36 10 d after PH). Data indicated that about 2/3 of the original residual hepatocytes entered S-phase in response to PH. Analysis of cluster size distribution at 24, 48, 96 h and 10 d after PH revealed that about half of the remnant hepatocytes completed at least 2 cell cycles. Average size of hepatocytes increased at 24 h (248.50 μm 2 ± 7.82 μm 2 , P = 0.0015), but returned to control values throughout the regenerative process (up to 10 d post-PH, 197.9 μm 2 ± 6.44 μm 2 , P = 0.11). A sizeable fraction of the remnant hepatocyte population does not participate actively in tissue mass restoration. Hyperplasia stands as the major mechanism contributing to liver mass restoration after PH, with hypertrophy playing a transient role in the process.

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